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Sunday, March 21, 2010 — 8:30 AM, Maryland
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Moderator:
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Sherif Zaki, Centers for Disease Control and Prevention, Atlanta, GA
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Disclosure:
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In accordance with ACCME guidelines regarding disclosure, the USCAP policy requires that faculty members who have a significant financial or other relationship with a commercial company, entity, or service (which will be discussed in this Symposium) must disclose this to attendees. The Academy also requires that speakers disclose any products that are not labeled for the use under discussion. The speakers listed below have indicated they have nothing to disclose.
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 Click here to view (or print) handout in single page format for the entire meeting.
Note: Any slides will be printed in color.
Click any blue title below to display and print the handout for individual lecture(s):
| 8:30 | Sepsis in Pregnancy - Pathogenesis, Routes of Entry, and Reasons for Mortality - Sebastian Lucas, King's College London School of Medicine, London, UK |
| 9:15 | Pathology and Pathogenesis of 2009 Pandemic H1N1 Influenza - Wun-Ju Shieh, Centers for Disease Control and Prevention, Atlanta, GA |
| 10:00 | Break |
| 10:30 | Pathogenesis of Rickettsial Diseases - David Walker, University of Texas Medical Branch, Galveston, TX |
| 11:15 | Update on Allergic Bronchopulmonary Aspergillosis - Dani S. Zander, Penn State University College of Medicine, Hershey, PA |
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Topics to be addressed were suggested and focus was established based on areas of perceived need for the enhanced education of all pathologists as determined by the expertise of the Program Committee members. to be important, this session also introduces key concepts in diagnosis and treatment of infectious diseases and understanding the pathologic and pathogenetic basis for various microbial infections (bacterial, fungal, viral, and rickettsial). The committee members addressed this goal by selecting speakers known to be highly competent pathologists who conduct primary research in the selected areas. The objective is to educate the attendees regarding pathological, histopathological responses, and pathogenetic mechanisms among infected persons with these diverse etiologic agents. |
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